RevGenetics: TA-65: 100 Unit Telomerase Activator
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SKU: TA65100u-30

TA-65: 100 Unit Telomerase Activator

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TA65 Lengthens Human Telomeres. Yes A 2016 Human Study Proves TA65 Lengthens Telomeres in Human Subjects (TA-65 Lengthens Telomeres). This Is The Only Product Tested On Humans And Proven To Lengthen Telomeres. We Believe TA-65 Is The Most Bioavailable Telomerase Activator Available Today As It Is Up To 50 Fold Better Than Anything Else We Have Seen. The product may be different than shown. *We have provided a this discounted price matched price today. It may change from one day to the next, please order today to save!.

Quantity:
$100.00

The TA-65 TA-65 Is BioenhancedFormula Is Simply 50x More Bioavailable Than Our Previously Recommended Telomerase Activation Products.

Not Only Will You Get The Best, But You Also Get The Lowest Prices, As We Were The First To Implement The TA-65 Low Price Guarantee! (Click To Read More)

The TA-65 product has been tested by RevGenetics on live cells (in Vitro). Recently RevGenetics met with TA Sciences in NY and were provided direct evidence that TA-65 Bioenhanced complex is 50x to 100x Better than our previously recommended products which had the same molecule.

We now believe the Bioenhanced TA-65 simply provides much higher absorption and telomerase activation than any others we have tested.

Why are we the best place to get TA-65? RevGenetics was the first RevGenetics and the only company who has independently tested TA-65®. RevGenetics was the first to provide TA-65 online (other than physicians), RevGenetics is the only company that has gone to New York to meet with TA Sciences to gather information about human PK studies, RevGenetics was one of the first companies to push for and get a lower priced $100 bottle of TA-65 for you and last but not least RevGenetics not only sells supplements, but we provide US Government labs material for research as well as give access to students to lab equipment to help them reach their PhDs.

Giving back to the community: When you purchase your products from RevGenetics, you help students, ongoing research in longevity, and the development of unique new products. Your initial shipment will take 1-2 weeks to arrive. We Are Not Only Trusted By Universities, But We Are Also Trusted by the BBB with an "A" rating.

Your Purchase Today Is Covered Through The BBB. RevGenetics is trusted by the BBB with an "A" rating for our excellent service. Why are we better? We are the only company who has independently tested TA-65® with human cells using the same methods used by UCLA. The new UCLA study has now been published by our researcher Dr. H. F. Valenzuela (Link). The new 2013 study proves that the product activates telomerase to a statistically significant degree in human cells. However we do not recommend TA-65 for an increase in longevity but just for it's concentrated Astragalus benefits.

According to other studies on TA-65 telomerase activation, telomerase:

• Lengthens Short Telomeres • Repairs DNA Damage • Rejuvenates Aging Immune Systems • Increases Bone Density • Improves Biomarkers that Decline with Age

RevGenetics Now Certifies TA-65®: To test the product, RevGenetics first purchased TA-65 anonymously, we did not receive free TA-65 for our lab tests. After the TA-65® telomerase product was lab tested with positive results, we decided to make it available to our customers. Because we believe that the TA-65® telomerase benefits can only be achieved through a 3 to 6 month use of the product, there are no returns or refunds on this product.

Have a question about this product? Please Read out TA-65 FAQ (TA-65 Frequently Asked Questions) What is Autoship? It is a special quantity discount for this item. By ordering using our recurring autoship plan, you effectively lock in the quantity discount price for future shipments and can cancel at anytime after the second shipment you receive from us. Yes, you can lock in this discount rate if you try our recurring autoship option, even if it's changed at a later date or we decide not to offer the quantity discount anymore. Our best recurring offer allows us to ship you 2 large 90 capsule bottles of TA-65 for $1000, that's only $500 a bottle!

No prescription or doctor approval is required for the sale of this product. 
The product is herbal and does not contain prescription ingredients.

Patented Technology

Please note: This has been RevGenetics Tested, Geron Patented. Purchase Your Personal TA-65® Telomerase Activator Today If an international order exceeds $650, we will break the order into multiple packages and invoices. RevGenetics will choose the best shipping method for your order, while international customers taxes, customs, duties are to be paid by the customer for all International orders. Recurring orders are charged every 30 days or every 90 days, however if you email us we can change this to charge you in a recurring time frame that is more convenient. If selecting a recurring order, you agree to accept at least two shipments before canceling further shipments. Please check with customs if this product can be shipped to your country as we don't offer refunds if your country prohibits this shipment into your country, or particular address. Purchase your TA-65 from RevGenetics today and help support independent research!

*The statements on this page have not been evaluated by the Food and Drug Administration. This supplement is not intended to diagnose, treat, cure or prevent any disease. TA-65® Is a registered trademark of Telomerase Activation Sciences, Inc. Buy TA-65® at RevGenetics today to support research and new product development. The Top Source For Your TA-65 Online. 

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Nicotinamide Mononucleotide NAD+ And Other Study References:

  1. Detection and pharmacological modulation of nicotinamide mononucleotide (NMN) in vitro and in vivo - (Formentini, 2009)
  2. AMPK regulates energy expenditure by modulating NAD+ metabolism and SIRT1 activity - (Cato, 2009)
  3. A possibility of nutriceuticals as an anti-aging intervention: activation of sirtuins by promoting mammalian NAD biosynthesis - (Imai, 2010)
  4. NAD blocks high glucose induced mesangial hypertrophy via activation of the sirtuins-AMPK-mTOR pathway - (Zhuo, 2011)
  5. Nicotinamide Mononucleotide, a Key NAD+ Intermediate, Treats the Pathophysiology of Diet- and Age-Induced Diabetes in Mice - (Yoshino, 2011)
  6. The NAD (+) precursor nicotinamide riboside enhances oxidative metabolism and protects against high-fat diet-induced obesity - (Canto, 2012 )
  7. NAD⁺ repletion improves mitochondrial and stem cell function and enhances life span in mice. - (Zhang, 2016)
  8. Declining NAD+ Induces a Pseudohypoxic State Disrupting Nuclear-Mitochondrial Communication during Aging - (Gomes, Sinclair,2013)
  9. Nicotinamide mononucleotide, an intermediate of NAD+ synthesis, protects the heart from ischemia and repercussion - (Yamamoto, 2014)
  10. NAD+ and sirtuins in aging and disease - (Imai, 2014)
  11. Effective treatment of mitochondrial myopathy by nicotinamide riboside, a vitamin B3 - (Khan, 2014)
  12. Effect of nicotinamide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer’s disease-relevant murine model - (Long, 2015)
  13. NAD+ metabolism and the control of energy homeostasis – a balancing act between mitochondria and the nucleus - (Canto, 2015)
  14. NAD+ metabolism: Bioenergetics, signaling and manipulation for therapy  - (Yang, 2016)
  15. NAD+ replenishment improves lifespan and healthspan in ataxia telangiectasia models via mitophagy and DNA repair - (Fang, 2016)
  16. Nicotinamide riboside is uniquely and orally bioavailable in mice and humans - (Trammell, 2016)
  17. Nicotinamide riboside opposes type 2 diabetes and neuropathy in mice - (Trammell, 2016)
  18. β-Nicotinamide Mononucleotide, an Anti-Aging Candidate Compound, Is Retained in the Body for Longer than Nicotinamide in Rats - (Kawamura, 2016)
  19. The first human clinical study for NMN has started in Japan - (Tsubota, 2016)
  20. Nicotinamide mononucleotide protects against β-amyloid oligomer-induced cognitive impairment and neuronal death - (Wang, 2016)
  21. Head to Head Comparison of Short-Term Treatment with the NAD(+) Precursor Nicotinamide Mononucleotide (NMN) and 6 Weeks of Exercise in Obese Female Mice - (Uddin, 2016)
  22. Long-Term Administration of Nicotinamide Mononucleotide Mitigates Age-Associated Physiological Decline in Mice - (Mills, 2016)
  23. Nicotinamide mononucleotide supplementation reverses vascular dysfunction and oxidative stress with aging in mice - (de Picciotto, 2016)
  24. Nicotinamide mononucleotide inhibits JNK activation to reverse Alzheimer disease - (Yao, 2017)
  25. Nicotinamide mononucleotide requires SIRT3 to improve cardiac function and bioenergetics in a Friedreich’s ataxia cardiomyopathy model - (Martin, 2017)
  26. Nicotinamide Mononucleotide, an NAD+ Precursor, Rescues Age-Associated Susceptibility to AKI in a Sirtuin 1-Dependent Manner - (Guan, 2017)
  27. Nicotinamide mononucleotide attenuates brain injury after intracerebral hemorrhage by activating Nrf2/HO-1 signaling pathway - (Wei, 2017)
  28. Short-term administration of Nicotinamide Mononucleotide preserves cardiac mitochondrial homeostasis and prevents heart failure - (Zhang, 2017)
  29. Modulating NAD+ metabolism, from bench to bedside - (Auwerx, 2017)
  30. Aspects of Tryptophan and Nicotinamide Adenine Dinucleotide in Immunity: A New Twist in an Old Tale. - (Rodriguez, 2017)
  31. Vitamin B3 modulates mitochondrial vulnerability and prevents glaucoma in aged mice - (Williams, 2017)
  32. NAMPT-mediated NAD biosynthesis as the internal timing mechanism: In NAD+ World, time is running in its own way - (Poljsak, 2017)
  33. Effect of “Nicotinamide Mononucleotide” (NMN) on Cardiometabolic Function (NMN) - (Clinical In Process)
  34. The dynamic regulation of NAD metabolism in mitochondria - (Stein, 2012)
  35. Novel NAD+ metabolomic technologies and their applications to Nicotinamide Riboside interventions - (Trammel, 2016)
  36. Long-term moderate calorie restriction inhibits inflammation without impairing cell-mediated immunity: a randomized controlled trial in non-obese humans - (Meydayni, 2016)
  37. A high-fat, ketogenic diet induces a unique metabolic state in mice.  - (Kennedy, 2007)
  38. Ketone body metabolism and cardiovascular disease. - (Cotter, 2013)
  39. Ketone bodies as signaling metabolites - (Newman, 2014)
  40. The ketone metabolite β-hydroxybutyrate blocks NLRP3 inflammasome–mediated inflammatory disease - (Youm, 2015)
  41. The effect of the Spanish Ketogenic Mediterranean Diet on nonalcoholic fatty liver disease: a pilot study. - (Guisado, 2011)
  42. β-Hydroxybutyrate: A Signaling Metabolite in starvation response - (Morales, 2016)
  43. Physiological roles of ketone bodies as substrates and signals in mammalian tissues - (Robinson, 1980)
  44. Ketone bodies mimic the life span extending properties of caloric restriction  - (Veech, 2017)
  45. Novel ketone diet enhances physical and cognitive performance - (Murray, 2016)
  46. Mitochondrial biogenesis and increased uncoupling protein 1 in brown adipose tissue of mice fed a ketone ester diet. - (Study)
  47. Nutritional Ketosis Alters Fuel Preference and Thereby Endurance Performance in Athletes - (Cox, 2013)
  48. Neuroendocrine Factors in the Regulation of Inflammation: Excessive Adiposity and Calorie Restriction - (Fontana, 2009)
  49. Beta-adrenergic receptors are critical for weight loss but not for other metabolic adaptations to the consumption of a ketogenic diet in male mice - (August, 2017)
  50. A randomized trial of a low-carbohydrate diet for obesity - (Foster, 2003)
  51. β-Hydroxybutyrate suppresses inflammasome formation by ameliorating endoplasmic reticulum stress via AMPK activation - (Bae, 2016)
  52. The neuroprotective properties of calorie restriction, the ketogenic diet, and ketone bodies.  - (Maalouf, 2009)
  53. AMPK activation protects cells from oxidative stress‐induced senescence via autophagic flux restoration and intracellular NAD + elevation  - (Han, 2016)
  54. Regulation of AMP-activated protein kinase by natural and synthetic activators - (Hardie, 2015)
  55. Effects of Exhaustive Aerobic Exercise on Tryptophan-Kynurenine Metabolism in Trained Athletes  - (Strasser, 2016)
  56. PARP-1 inhibition increases mitochondrial metabolism through SIRT1 activation - (Bai, 2011)
  57. Carbohydrate restriction regulates the adaptive response to fasting - (Klein, 1992)
  58. Interventions to Slow Aging in Humans: Are We Ready? - (longo, 2015)
  59. Extending healthy life span–from yeast to humans - (longo, 2010)
  60. Dietary restriction with and without caloric restriction for healthy aging - (Lee, 2016)
  61. A Periodic Diet that Mimics Fasting Promotes Multi-System Regeneration, Enhanced Cognitive Performance, and Healthspan - (Longo, 2015)
  62. Diet mimicking fasting promotes regeneration and reduces autoimmunity and multiple sclerosis symptoms - (Longo, 2016)
  63. Resistance Exercise Training Alters Mitochondrial Function in Human Skeletal Muscle - (Porter, 2015)
  64. Ketogenic Diet Reduces Midlife Mortality and Improves Memory in Aging Mice  - (Newman, 2017)
  65. The NAD(+)/sirtuin pathway modulates longevity through activation of mitochondrial UPR and FOXO signaling - (Mouchiroud, 2013)
  66. NAMPT- mediated NAD(+) biosynthesis is essential for vision in mice - (Lin, 2016)
  67. NAD+ replenishment improves lifespan and healthspan in ataxia telangiectasia models via mitophagy and DNA repair - (Fang, 2016)
  68. Inhibiting poly ADP-ribosylation increases fatty acid oxidation and protects against fatty liver disease - (Gariani, 2017 )
  69. Interdependence of AMPK and SIRT1 for metabolic adaptation to fasting and exercise in skeletal muscle - (Canto, 2010)
  70. The NAD (+) precursor nicotinamide riboside enhances oxidative metabolism and protects against high-fat diet-induced obesity - (Canto, 2012)
  71. Nicotinamide riboside is uniquely and orally bioavailable in mice and humans - (Trammell, 2016)
  72. Nicotinamide riboside opposes type 2 diabetes and neuropathy in mice - (Trammell, 2016)
  73. Dietary leucine stimulates SIRT1 signaling through activation of AMPK - (Hongliang, 2012)
  74. Effective treatment of mitochondrial myopathy by nicotinamide riboside, a vitamin B3 - (Khan, 2014)
  75. NAD blocks high glucose induced mesangial hypertrophy via activation of the sirtuins-AMPK-mTOR pathway - (Zhuo, 2011)
  76. The effect of different exercise regimens on mitochondrial biogenesis and performance - (Philander, 2014)
  77. Dietary proanthocyanidins boost hepatic NAD+ metabolism and SIRT1 expression and activity in a dose-dependent manner in healthy rats - (Aragon’s, 2016)
  78. NAD+ Deficits in Age-Related Diseases and Cancer - (Garrido, 2017)
  79. Anti-diabetic and anti-lipidemic effects of chlorogenic acid are mediated by ampk activation - (Ong, 2013)
  80. Chlorogenic Acid Improves Late Diabetes through Adiponectin Receptor Signaling Pathways in db/db Mice - (Chang, 2015)
  81. Adenosine Monophosphate (AMP)-Activated Protein Kinase: A New Target for Nutraceutical Compounds - (Marin-Aguilar, 2017)
  82. The Effects of Ramadan Fasting on Body Composition, Blood Pressure, Glucose Metabolism, and Markers of Inflammation in NAFLD Patients: An Observational Trial - (Mazidi, 2014)
  83. Comparative effects of carbohydrate versus fat restriction on metabolic profiles, biomarkers of inflammation and oxidative stress in overweight patients with Type 2 diabetic and coronary heart disease: A randomized clinical trial. - (Raygan, 2016)
  84. Normal fasting plasma glucose and risk of type 2 diabetes diagnosis - (Nichols, 2008)
  85. Are We All Pre-Diabetic? - (Stokel,2016)
  86. Hepatic NAD+ deficiency as a therapeutic target for non-alcoholic fatty liver disease in aging - (Zhou, 2016)
  87. Effect of exercise intensity on post-exercise oxygen consumption and heart rate recovery - (Mann,2014)
  88. A 45-minute vigorous exercise bout increases metabolic rate for 14 hours - (Knab,2011)
  89. Effects of high-intensity resistance training on untrained older men. II. Muscle fiber characteristics and nuclei-cytoplasmic relationships - (Gerontol, 2000)
  90. Ketogenic Diet Reduces Midlife Mortality and Improves Memory in Aging Mice - (Newman, 2017)
  91. A Ketogenic Diet Extends Longevity and Healthspan in Adult Mice - (Roberts, 2017)
  92. NK cells link obesity-induced adipose stress to inflammation and insulin resistance - (Wensveen, 2015)
  93. The “Big Bang” in obese fat: Events initiating obesity-induced adipose tissue inflammation - (Wensveen, 2015)
  94. The impact of the Standard American Diet in rats: Effects on behavior, physiology and recovery from inflammatory injury - (Totsch, 2017)
  95. Bioenergetic state regulates innate inflammatory responses through the transcriptional co-repressor CtBP - (Shen, 2017)
  96. The Ketogenic Diet as a Treatment Paradigm for Diverse Neurological Disorders - (Stafstrom, 2012)
  97. Loss of NAD Homeostasis Leads to Progressive and Reversible Degeneration of Skeletal Muscle - (Fredrick 2016)
  98. Digestion and absorption of NAD by the small intestine of the rat - (Henderson, 1983)
  99. Effects of a wide range of dietary nicotinamide riboside (NR) concentrations on metabolic flexibility and white adipose tissue (WAT) of mice fed a mildly obesogenic diet - (Shi, 2017)
  100. Discoveries of nicotinamide riboside as a nutrient and conserved NRK genes establish a Preiss-Handler independent route to NAD+ in fungi and humans  - (Brenner, 2004)
  101. Nampt Expression Decreases Age-Related Senescence in Rat Bone Marrow Mesenchymal Stem Cells by Targeting Sirt1 - (Ma, 2017)

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