RG-Cell: Stem Cell Serum For Skin Care
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SKU: RGCELL-5ML

RG-Cell: Stem Cell Serum For Skin Care

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RG-Cell: Stem Cell Serum For Skin Care RGCell Replaces Many Cosmetics TimeSlip RG-CELL ® Concentrated Restorative Skin Serum is the hottest, breakthrough, anti-aging, skin treatment method cosmeceutical to reach the marketplace. It uses an one-of-a-kind exclusive fusion of stem cell activators designed to preserve your skin and clearly fight damage at the cell level.

Each and every hard-working factor within RG-Cell ® Concentrated Restorative Serum teams up synergistically, drawing cellular action for invigorated collagen, elastin, laminins and various body skin health proteins. Just what we end up with, is a solution that revitalizes, mends and tones up mistreated skin.

RGCELL's Key Rh-Epidermal Growth Factor (EGF) attained by genetic engineering systems totally equivalent to all-natural EGF, recognized as a "ELEGANCE FACTOR", increases and manages stem cell spreading. Because of its recombinant systems it is absolutely safe compared with various other products that utilize human derived stem cells. The advancement of EGF earned Stanley Cohen of Vanderbilt University a Nobel Prize in Physiology and Medicine in 1986. Rh-EGF has now develop into the very first choice to do away with wrinkle without the unfavorable reactions of Botox Treatment option. RG-Cell Scientifically Tried and tested Anti-Aging and Anti-Wrinkling Skin Care This is definitely Scientifically Confirmed Skin Health Care!

The outstanding final results of 2 new research investigations validate conveniences of TimeSlip RG-CELL ® Cosmeceuticals showing that RG-CELL Concentrated Restorative Serum functions at the cellular level, notably increasing Collagen Type I and Collagen Type III Expression in in-vitro fibroblasts culture. Stem Cell Skin Care From RG-Cell Information on how it Functions RG-Cell ® is the initial cosmeceutical with the power to switch on aged and inactive stem cells in an all-natural way and refresh your skin to a much better, more youthful appearance. RG-Cell ® Concentrated Restorative Serum includes EGF a cellular activator, and skin healthy protein which instructs aging skin to boost up its repair method. It's topical application embodies the future of scientific skin health care and is founded on 10 years of biotechnology experimentation.

RG-Cell ® Concentrated Restorative Serum additionally showcases an unique and exclusive blend of stem cell activators each selected as a result of their scientifically confirmed ability to tap into our skin's stem cells. Revitalizing your latent stem cells and enlisting the use of your own individual skin cells is certainly the most reliable approach for skin revitalization as well as remodeling. This specific remarkable strategy promotes fibroblast formation of collagen, thus enhancing skin suppleness and suppleness.

The conclusion is, a renewed skin structure, a soft tone, decreased redness and a sleeker, silkier, much more vivid and more youthful looking skin. Stem Cell Skin Care Collagen Generation Instilled utilizing a fusion of particularly reliable anti-oxidants and the moisture capturing Hyaluronic Acid, RG-Cell ® stem cell skin care serum also encourages to revitalize radiance and suppleness and to boost the skin's immune capability to protect from future aging.

Note: No Artificial Colourings-- No Artificial Fragrances-- No Mineral Oil-- No Parabens-- No Animal By-products-- No Bleaching & Deodorizing agents-- No harmful ingredients

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Nicotinamide Mononucleotide NAD+ And Other Study References:

  1. Detection and pharmacological modulation of nicotinamide mononucleotide (NMN) in vitro and in vivo - (Formentini, 2009)
  2. AMPK regulates energy expenditure by modulating NAD+ metabolism and SIRT1 activity - (Cato, 2009)
  3. A possibility of nutriceuticals as an anti-aging intervention: activation of sirtuins by promoting mammalian NAD biosynthesis - (Imai, 2010)
  4. NAD blocks high glucose induced mesangial hypertrophy via activation of the sirtuins-AMPK-mTOR pathway - (Zhuo, 2011)
  5. Nicotinamide Mononucleotide, a Key NAD+ Intermediate, Treats the Pathophysiology of Diet- and Age-Induced Diabetes in Mice - (Yoshino, 2011)
  6. The NAD (+) precursor nicotinamide riboside enhances oxidative metabolism and protects against high-fat diet-induced obesity - (Canto, 2012 )
  7. NAD⁺ repletion improves mitochondrial and stem cell function and enhances life span in mice. - (Zhang, 2016)
  8. Declining NAD+ Induces a Pseudohypoxic State Disrupting Nuclear-Mitochondrial Communication during Aging - (Gomes, Sinclair,2013)
  9. Nicotinamide mononucleotide, an intermediate of NAD+ synthesis, protects the heart from ischemia and repercussion - (Yamamoto, 2014)
  10. NAD+ and sirtuins in aging and disease - (Imai, 2014)
  11. Effective treatment of mitochondrial myopathy by nicotinamide riboside, a vitamin B3 - (Khan, 2014)
  12. Effect of nicotinamide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer’s disease-relevant murine model - (Long, 2015)
  13. NAD+ metabolism and the control of energy homeostasis – a balancing act between mitochondria and the nucleus - (Canto, 2015)
  14. NAD+ metabolism: Bioenergetics, signaling and manipulation for therapy  - (Yang, 2016)
  15. NAD+ replenishment improves lifespan and healthspan in ataxia telangiectasia models via mitophagy and DNA repair - (Fang, 2016)
  16. Nicotinamide riboside is uniquely and orally bioavailable in mice and humans - (Trammell, 2016)
  17. Nicotinamide riboside opposes type 2 diabetes and neuropathy in mice - (Trammell, 2016)
  18. β-Nicotinamide Mononucleotide, an Anti-Aging Candidate Compound, Is Retained in the Body for Longer than Nicotinamide in Rats - (Kawamura, 2016)
  19. The first human clinical study for NMN has started in Japan - (Tsubota, 2016)
  20. Nicotinamide mononucleotide protects against β-amyloid oligomer-induced cognitive impairment and neuronal death - (Wang, 2016)
  21. Head to Head Comparison of Short-Term Treatment with the NAD(+) Precursor Nicotinamide Mononucleotide (NMN) and 6 Weeks of Exercise in Obese Female Mice - (Uddin, 2016)
  22. Long-Term Administration of Nicotinamide Mononucleotide Mitigates Age-Associated Physiological Decline in Mice - (Mills, 2016)
  23. Nicotinamide mononucleotide supplementation reverses vascular dysfunction and oxidative stress with aging in mice - (de Picciotto, 2016)
  24. Nicotinamide mononucleotide inhibits JNK activation to reverse Alzheimer disease - (Yao, 2017)
  25. Nicotinamide mononucleotide requires SIRT3 to improve cardiac function and bioenergetics in a Friedreich’s ataxia cardiomyopathy model - (Martin, 2017)
  26. Nicotinamide Mononucleotide, an NAD+ Precursor, Rescues Age-Associated Susceptibility to AKI in a Sirtuin 1-Dependent Manner - (Guan, 2017)
  27. Nicotinamide mononucleotide attenuates brain injury after intracerebral hemorrhage by activating Nrf2/HO-1 signaling pathway - (Wei, 2017)
  28. Short-term administration of Nicotinamide Mononucleotide preserves cardiac mitochondrial homeostasis and prevents heart failure - (Zhang, 2017)
  29. Modulating NAD+ metabolism, from bench to bedside - (Auwerx, 2017)
  30. Aspects of Tryptophan and Nicotinamide Adenine Dinucleotide in Immunity: A New Twist in an Old Tale. - (Rodriguez, 2017)
  31. Vitamin B3 modulates mitochondrial vulnerability and prevents glaucoma in aged mice - (Williams, 2017)
  32. NAMPT-mediated NAD biosynthesis as the internal timing mechanism: In NAD+ World, time is running in its own way - (Poljsak, 2017)
  33. Effect of “Nicotinamide Mononucleotide” (NMN) on Cardiometabolic Function (NMN) - (Clinical In Process)
  34. The dynamic regulation of NAD metabolism in mitochondria - (Stein, 2012)
  35. Novel NAD+ metabolomic technologies and their applications to Nicotinamide Riboside interventions - (Trammel, 2016)
  36. Long-term moderate calorie restriction inhibits inflammation without impairing cell-mediated immunity: a randomized controlled trial in non-obese humans - (Meydayni, 2016)
  37. A high-fat, ketogenic diet induces a unique metabolic state in mice.  - (Kennedy, 2007)
  38. Ketone body metabolism and cardiovascular disease. - (Cotter, 2013)
  39. Ketone bodies as signaling metabolites - (Newman, 2014)
  40. The ketone metabolite β-hydroxybutyrate blocks NLRP3 inflammasome–mediated inflammatory disease - (Youm, 2015)
  41. The effect of the Spanish Ketogenic Mediterranean Diet on nonalcoholic fatty liver disease: a pilot study. - (Guisado, 2011)
  42. β-Hydroxybutyrate: A Signaling Metabolite in starvation response - (Morales, 2016)
  43. Physiological roles of ketone bodies as substrates and signals in mammalian tissues - (Robinson, 1980)
  44. Ketone bodies mimic the life span extending properties of caloric restriction  - (Veech, 2017)
  45. Novel ketone diet enhances physical and cognitive performance - (Murray, 2016)
  46. Mitochondrial biogenesis and increased uncoupling protein 1 in brown adipose tissue of mice fed a ketone ester diet. - (Study)
  47. Nutritional Ketosis Alters Fuel Preference and Thereby Endurance Performance in Athletes - (Cox, 2013)
  48. Neuroendocrine Factors in the Regulation of Inflammation: Excessive Adiposity and Calorie Restriction - (Fontana, 2009)
  49. Beta-adrenergic receptors are critical for weight loss but not for other metabolic adaptations to the consumption of a ketogenic diet in male mice - (August, 2017)
  50. A randomized trial of a low-carbohydrate diet for obesity - (Foster, 2003)
  51. β-Hydroxybutyrate suppresses inflammasome formation by ameliorating endoplasmic reticulum stress via AMPK activation - (Bae, 2016)
  52. The neuroprotective properties of calorie restriction, the ketogenic diet, and ketone bodies.  - (Maalouf, 2009)
  53. AMPK activation protects cells from oxidative stress‐induced senescence via autophagic flux restoration and intracellular NAD + elevation  - (Han, 2016)
  54. Regulation of AMP-activated protein kinase by natural and synthetic activators - (Hardie, 2015)
  55. Effects of Exhaustive Aerobic Exercise on Tryptophan-Kynurenine Metabolism in Trained Athletes  - (Strasser, 2016)
  56. PARP-1 inhibition increases mitochondrial metabolism through SIRT1 activation - (Bai, 2011)
  57. Carbohydrate restriction regulates the adaptive response to fasting - (Klein, 1992)
  58. Interventions to Slow Aging in Humans: Are We Ready? - (longo, 2015)
  59. Extending healthy life span–from yeast to humans - (longo, 2010)
  60. Dietary restriction with and without caloric restriction for healthy aging - (Lee, 2016)
  61. A Periodic Diet that Mimics Fasting Promotes Multi-System Regeneration, Enhanced Cognitive Performance, and Healthspan - (Longo, 2015)
  62. Diet mimicking fasting promotes regeneration and reduces autoimmunity and multiple sclerosis symptoms - (Longo, 2016)
  63. Resistance Exercise Training Alters Mitochondrial Function in Human Skeletal Muscle - (Porter, 2015)
  64. Ketogenic Diet Reduces Midlife Mortality and Improves Memory in Aging Mice  - (Newman, 2017)
  65. The NAD(+)/sirtuin pathway modulates longevity through activation of mitochondrial UPR and FOXO signaling - (Mouchiroud, 2013)
  66. NAMPT- mediated NAD(+) biosynthesis is essential for vision in mice - (Lin, 2016)
  67. NAD+ replenishment improves lifespan and healthspan in ataxia telangiectasia models via mitophagy and DNA repair - (Fang, 2016)
  68. Inhibiting poly ADP-ribosylation increases fatty acid oxidation and protects against fatty liver disease - (Gariani, 2017 )
  69. Interdependence of AMPK and SIRT1 for metabolic adaptation to fasting and exercise in skeletal muscle - (Canto, 2010)
  70. The NAD (+) precursor nicotinamide riboside enhances oxidative metabolism and protects against high-fat diet-induced obesity - (Canto, 2012)
  71. Nicotinamide riboside is uniquely and orally bioavailable in mice and humans - (Trammell, 2016)
  72. Nicotinamide riboside opposes type 2 diabetes and neuropathy in mice - (Trammell, 2016)
  73. Dietary leucine stimulates SIRT1 signaling through activation of AMPK - (Hongliang, 2012)
  74. Effective treatment of mitochondrial myopathy by nicotinamide riboside, a vitamin B3 - (Khan, 2014)
  75. NAD blocks high glucose induced mesangial hypertrophy via activation of the sirtuins-AMPK-mTOR pathway - (Zhuo, 2011)
  76. The effect of different exercise regimens on mitochondrial biogenesis and performance - (Philander, 2014)
  77. Dietary proanthocyanidins boost hepatic NAD+ metabolism and SIRT1 expression and activity in a dose-dependent manner in healthy rats - (Aragon’s, 2016)
  78. NAD+ Deficits in Age-Related Diseases and Cancer - (Garrido, 2017)
  79. Anti-diabetic and anti-lipidemic effects of chlorogenic acid are mediated by ampk activation - (Ong, 2013)
  80. Chlorogenic Acid Improves Late Diabetes through Adiponectin Receptor Signaling Pathways in db/db Mice - (Chang, 2015)
  81. Adenosine Monophosphate (AMP)-Activated Protein Kinase: A New Target for Nutraceutical Compounds - (Marin-Aguilar, 2017)
  82. The Effects of Ramadan Fasting on Body Composition, Blood Pressure, Glucose Metabolism, and Markers of Inflammation in NAFLD Patients: An Observational Trial - (Mazidi, 2014)
  83. Comparative effects of carbohydrate versus fat restriction on metabolic profiles, biomarkers of inflammation and oxidative stress in overweight patients with Type 2 diabetic and coronary heart disease: A randomized clinical trial. - (Raygan, 2016)
  84. Normal fasting plasma glucose and risk of type 2 diabetes diagnosis - (Nichols, 2008)
  85. Are We All Pre-Diabetic? - (Stokel,2016)
  86. Hepatic NAD+ deficiency as a therapeutic target for non-alcoholic fatty liver disease in aging - (Zhou, 2016)
  87. Effect of exercise intensity on post-exercise oxygen consumption and heart rate recovery - (Mann,2014)
  88. A 45-minute vigorous exercise bout increases metabolic rate for 14 hours - (Knab,2011)
  89. Effects of high-intensity resistance training on untrained older men. II. Muscle fiber characteristics and nuclei-cytoplasmic relationships - (Gerontol, 2000)
  90. Ketogenic Diet Reduces Midlife Mortality and Improves Memory in Aging Mice - (Newman, 2017)
  91. A Ketogenic Diet Extends Longevity and Healthspan in Adult Mice - (Roberts, 2017)
  92. NK cells link obesity-induced adipose stress to inflammation and insulin resistance - (Wensveen, 2015)
  93. The “Big Bang” in obese fat: Events initiating obesity-induced adipose tissue inflammation - (Wensveen, 2015)
  94. The impact of the Standard American Diet in rats: Effects on behavior, physiology and recovery from inflammatory injury - (Totsch, 2017)
  95. Bioenergetic state regulates innate inflammatory responses through the transcriptional co-repressor CtBP - (Shen, 2017)
  96. The Ketogenic Diet as a Treatment Paradigm for Diverse Neurological Disorders - (Stafstrom, 2012)
  97. Loss of NAD Homeostasis Leads to Progressive and Reversible Degeneration of Skeletal Muscle - (Fredrick 2016)
  98. Digestion and absorption of NAD by the small intestine of the rat - (Henderson, 1983)
  99. Effects of a wide range of dietary nicotinamide riboside (NR) concentrations on metabolic flexibility and white adipose tissue (WAT) of mice fed a mildly obesogenic diet - (Shi, 2017)
  100. Discoveries of nicotinamide riboside as a nutrient and conserved NRK genes establish a Preiss-Handler independent route to NAD+ in fungi and humans  - (Brenner, 2004)
  101. Nampt Expression Decreases Age-Related Senescence in Rat Bone Marrow Mesenchymal Stem Cells by Targeting Sirt1 - (Ma, 2017)

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