RevGenetics: MetaCurcumin Maximum Strength: Super Curcumin In Liquid Capsules
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SKU: MCurcumin-Caps-60

MetaCurcumin Super Curcumin In Liquid Capsules

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ORDER TODAY AND SAVE 50% OFF WHEN BUYING 10 BOTTLES60 Liquid Capsules, Non-GMO, No Gluten, No Wheat, No Sugar, Dairy Free & 24 Hour Support As Curcumin Has Been Found After 24 Hours In The Plasma Of People Who Take This Liquid Curcumin. Our Guarantee: If Our Turmeric Super Curcumin Doesn't Beat Yours, Simply Return It Within 30 Days For A Full Refund Of Your Purchase. This Product Has Been Mentioned In This Alzheimers Video: (Micelle Curcumin Vs Alzheimers)*. The MetaCurcumin Maximum Strength Liquid Capsules Contains The Most Powerful Liquid Turmeric Super Curcumin In The World In Easy To Swallow Liquid Capsules And We Have Now Added Extra Curcuminoids That You Can See In Every Capsule.

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The MetaCurcumin micelle curcumin human bioavailability study outperformed the human studies done for whey curcumin complex, the Sabinsa C3 ® complex study, Longvida ® study, BCM95 ® study, LEF's Super Biocurcumin ® Study, Meriva ® Study and even those studies that use BioPerine ® and Black pepper. This product has Micelle Super Curcumin and in this new formula we have added additional Curcuminoids to make this a powerful Full Spectrum Turmeric Liquid Capsule. What about Liposome curcumin? Yes MetaCurcumin beats that as well. We are so sure MetaCurcumin will beat your turmeric curcumin that we provide a money back guarantee if it doesn't. 1 serving of MetaCurcumin Maximum Strength is as powerful as taking up to 23.2 grams of standard curcumin (Based On Women In University Study). MetaCurcumin micelle curcumin beats all other tested super curcumin products (including liposome curcumin products) we have seen. The charts below compares MetaCurcumin against popular curcumin products and material sold. MetaCurcumin micelle turmeric curcumin easily beats all of those seen in both bioavailability and speed of absorption. Regular curcumin is also usually not found after 24 hours in plasma because it has been excreted, however we have seen that some MetaCurcumin is still found in plasma even after 24 hours. This is the only product we have seen that does this to a significant degree

The charts below compares MetaCurcumin against popular curcumin products and material sold, which MetaCurcumin beats easily in both bioavailability and speed of absorption. MetaCurcumin Absorption Chart With our low price and estimated 30 servings per bottle, we believe it is the most affordable and powerful liquid capsule micelle curcumin product available today!

This enhanced bioavailability and sustained retention time in the body has been confirmed by a human scientific study on curcumin absorption.

Each Serving Of MetaCurcumin Maximum Strength Provides The Bioavailability Of Up To 23.2 Grams In Women Of Standard Curcumin. In addition, by maximizing the amount of liquid curcumin in each liquid capsule we are maximizing the amount of MetaCurcumin you get along with the maximum super curcumin absorption we provide your body. MetaCurcumin offers one of the best sources of bioavailable curcumin on earth, and now we have added extra curcuminoids to make this a full spectrum turmeric curcumin capsule.

We suggest taking 1 serving of MetaCurcumin Maximum Strength for every serving of pure organic resveratrol you take (or for every 250mg of micronized resveratrol powder you consume). Their is no taste as you are getting everything in a liquid capsule.
MetaCurcumin Speed Chart

MetaCurcumin Technology: The technology produces the highest absorption at the fastest speed of absorption, for curcumin available, all while using our maximum strength liquid capsules. MetaCurcumin Maximum Strength: Is produced by creating micelles on a nano scale that protect the ultra sheared nano particles of curcumin. These nano-sized micelles form a protective barrier around nano particles of curcumin and increase it's absorption dramatically, up to 277x in women!

The Micelle formation is essential for the absorption of fat-soluble vitamins and complicated lipids within the human body.With this technology, we now make the most powerful super curcumin available to you here at RevGenetics. This product does not use the black pepper extract or piperine to avoid issues with folks who need to avoid it.

What is Super Curcumin for? We recommend it to help fight "Inflammaging" and as one of the interventions in the aging, however, most know that curcumin has the following benefits:

Health Benefits at a Glance 1- About 200 times more absorbable than conventional curcumin supplements 2- WhoMetaCurcumin Nano Curcumin Technologyle-body health and wellness benefits for virtually every organ system 3- Suppresses inflammatory aspects like NF-kappaB protein molecule 4- Encourages healthy immune system function 5- Helps preserve healthy digestive capability Excellent nutrient for whole body system health Curcumin extract continues to excite scientists with its remarkable and many-faceted health and wellness benefits.

A component of ancient Ayurvedic medicine, curcumin extract restricts inflammatory factors in the body system. It also helps support digestive functionality, maintains immune system health, and more.

Curcumin inhibits overexpression of NF-kappB inflammatories and Curcumin encourages your body's healthy response to inflammation by inhibiting crucial inflammatory factors such as NF-kappaB, a protein molecule that acts like an "on-switch" in genes that govern the body's pro-inflammatory responses. Curcumin has been shown to apply potent inhibitory effects on NF-kappaB activation. Curcumin also inhibits the metabolism of arachidonic acid, as well as the activities of cyclooxygenase, lipoxygenase, and cytokines (interleukins and tumor necrosis factors).

Other clinical trials suggest that curcumin can promote healthy bowel function and joint health. Immune system and brain function health Some research studies indicate that curcumin can help preserve normal, healthy platelet function. Curcumin extract encourages immune system health by hindering histamine release from mast cells. Curcumin can also benefit your mind: In some studies, curcumin supported healthy brain function and offered neuro-protective benefits. Pancreas support and free radical protection Curcumin supports pancreatic function by promoting pancreatic islet health.

Curcumin's multifaceted benefits even include antioxidant protection and hormone balance support. A research study indicates curcumin can help protect against estrogen-mimicking chemicals, help shield against free radicals, and encourage normal cell cycle growth.

MetaCurcumin the worlds highest absorption curcumin. While it's hard to get adequate levels of curcumin into the bloodstream, MetaCurcumin utilizes special Micelle technology to get close to 277 times more of the super curcumin compound into a woman's bloodstream than standard curcumin supplements.

Customer Questions

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0
Q: Does this product have any artificial colors?

A: Not anymore. We used to use capsules with a trace of food coloring baked right into the capsule. But we have now transitioned to a clear capsule that contains no food coloring.

1
Q: The capsules are not solid orange anymore, why is that?

A: We used to use capsules with a trace of food coloring that was baked right into the capsule. This made them orange. We now use capsules without food coloring and are transparent.

2
Q: I can see powder inside the capsules along with the liquid, what is that?

A: We have added additional Curcuminoids to our formulation. The Micelle liquid curcumin is the pure liquid you see in the capsules, while the powder is the extra curcuminoids we have added. The additional curcuminoids can be seen in the transparent capsules as a light powder.

3
Q: What is your recommended dosing to help with inflammation for someone with a serious illness such as metastatic cancer ?

A: We apologize that we can't answer this questions. Currently, the FDA prohibits us from providing medical advice.

4
Q: Should MetaCurcumin be taken on empty stomach or with a meal?

A: MetaCurcumin Our Super Curcumin should be taken on an empty stomach or right before a meal. I hope this helps.

Nicotinamide Mononucleotide NAD+ And Other Study References:

  1. Detection and pharmacological modulation of nicotinamide mononucleotide (NMN) in vitro and in vivo - (Formentini, 2009)
  2. AMPK regulates energy expenditure by modulating NAD+ metabolism and SIRT1 activity - (Cato, 2009)
  3. A possibility of nutriceuticals as an anti-aging intervention: activation of sirtuins by promoting mammalian NAD biosynthesis - (Imai, 2010)
  4. NAD blocks high glucose induced mesangial hypertrophy via activation of the sirtuins-AMPK-mTOR pathway - (Zhuo, 2011)
  5. Nicotinamide Mononucleotide, a Key NAD+ Intermediate, Treats the Pathophysiology of Diet- and Age-Induced Diabetes in Mice - (Yoshino, 2011)
  6. The NAD (+) precursor nicotinamide riboside enhances oxidative metabolism and protects against high-fat diet-induced obesity - (Canto, 2012 )
  7. NAD⁺ repletion improves mitochondrial and stem cell function and enhances life span in mice. - (Zhang, 2016)
  8. Declining NAD+ Induces a Pseudohypoxic State Disrupting Nuclear-Mitochondrial Communication during Aging - (Gomes, Sinclair,2013)
  9. Nicotinamide mononucleotide, an intermediate of NAD+ synthesis, protects the heart from ischemia and repercussion - (Yamamoto, 2014)
  10. NAD+ and sirtuins in aging and disease - (Imai, 2014)
  11. Effective treatment of mitochondrial myopathy by nicotinamide riboside, a vitamin B3 - (Khan, 2014)
  12. Effect of nicotinamide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer’s disease-relevant murine model - (Long, 2015)
  13. NAD+ metabolism and the control of energy homeostasis – a balancing act between mitochondria and the nucleus - (Canto, 2015)
  14. NAD+ metabolism: Bioenergetics, signaling and manipulation for therapy  - (Yang, 2016)
  15. NAD+ replenishment improves lifespan and healthspan in ataxia telangiectasia models via mitophagy and DNA repair - (Fang, 2016)
  16. Nicotinamide riboside is uniquely and orally bioavailable in mice and humans - (Trammell, 2016)
  17. Nicotinamide riboside opposes type 2 diabetes and neuropathy in mice - (Trammell, 2016)
  18. β-Nicotinamide Mononucleotide, an Anti-Aging Candidate Compound, Is Retained in the Body for Longer than Nicotinamide in Rats - (Kawamura, 2016)
  19. The first human clinical study for NMN has started in Japan - (Tsubota, 2016)
  20. Nicotinamide mononucleotide protects against β-amyloid oligomer-induced cognitive impairment and neuronal death - (Wang, 2016)
  21. Head to Head Comparison of Short-Term Treatment with the NAD(+) Precursor Nicotinamide Mononucleotide (NMN) and 6 Weeks of Exercise in Obese Female Mice - (Uddin, 2016)
  22. Long-Term Administration of Nicotinamide Mononucleotide Mitigates Age-Associated Physiological Decline in Mice - (Mills, 2016)
  23. Nicotinamide mononucleotide supplementation reverses vascular dysfunction and oxidative stress with aging in mice - (de Picciotto, 2016)
  24. Nicotinamide mononucleotide inhibits JNK activation to reverse Alzheimer disease - (Yao, 2017)
  25. Nicotinamide mononucleotide requires SIRT3 to improve cardiac function and bioenergetics in a Friedreich’s ataxia cardiomyopathy model - (Martin, 2017)
  26. Nicotinamide Mononucleotide, an NAD+ Precursor, Rescues Age-Associated Susceptibility to AKI in a Sirtuin 1-Dependent Manner - (Guan, 2017)
  27. Nicotinamide mononucleotide attenuates brain injury after intracerebral hemorrhage by activating Nrf2/HO-1 signaling pathway - (Wei, 2017)
  28. Short-term administration of Nicotinamide Mononucleotide preserves cardiac mitochondrial homeostasis and prevents heart failure - (Zhang, 2017)
  29. Modulating NAD+ metabolism, from bench to bedside - (Auwerx, 2017)
  30. Aspects of Tryptophan and Nicotinamide Adenine Dinucleotide in Immunity: A New Twist in an Old Tale. - (Rodriguez, 2017)
  31. Vitamin B3 modulates mitochondrial vulnerability and prevents glaucoma in aged mice - (Williams, 2017)
  32. NAMPT-mediated NAD biosynthesis as the internal timing mechanism: In NAD+ World, time is running in its own way - (Poljsak, 2017)
  33. Effect of “Nicotinamide Mononucleotide” (NMN) on Cardiometabolic Function (NMN) - (Clinical In Process)
  34. The dynamic regulation of NAD metabolism in mitochondria - (Stein, 2012)
  35. Novel NAD+ metabolomic technologies and their applications to Nicotinamide Riboside interventions - (Trammel, 2016)
  36. Long-term moderate calorie restriction inhibits inflammation without impairing cell-mediated immunity: a randomized controlled trial in non-obese humans - (Meydayni, 2016)
  37. A high-fat, ketogenic diet induces a unique metabolic state in mice.  - (Kennedy, 2007)
  38. Ketone body metabolism and cardiovascular disease. - (Cotter, 2013)
  39. Ketone bodies as signaling metabolites - (Newman, 2014)
  40. The ketone metabolite β-hydroxybutyrate blocks NLRP3 inflammasome–mediated inflammatory disease - (Youm, 2015)
  41. The effect of the Spanish Ketogenic Mediterranean Diet on nonalcoholic fatty liver disease: a pilot study. - (Guisado, 2011)
  42. β-Hydroxybutyrate: A Signaling Metabolite in starvation response - (Morales, 2016)
  43. Physiological roles of ketone bodies as substrates and signals in mammalian tissues - (Robinson, 1980)
  44. Ketone bodies mimic the life span extending properties of caloric restriction  - (Veech, 2017)
  45. Novel ketone diet enhances physical and cognitive performance - (Murray, 2016)
  46. Mitochondrial biogenesis and increased uncoupling protein 1 in brown adipose tissue of mice fed a ketone ester diet. - (Study)
  47. Nutritional Ketosis Alters Fuel Preference and Thereby Endurance Performance in Athletes - (Cox, 2013)
  48. Neuroendocrine Factors in the Regulation of Inflammation: Excessive Adiposity and Calorie Restriction - (Fontana, 2009)
  49. Beta-adrenergic receptors are critical for weight loss but not for other metabolic adaptations to the consumption of a ketogenic diet in male mice - (August, 2017)
  50. A randomized trial of a low-carbohydrate diet for obesity - (Foster, 2003)
  51. β-Hydroxybutyrate suppresses inflammasome formation by ameliorating endoplasmic reticulum stress via AMPK activation - (Bae, 2016)
  52. The neuroprotective properties of calorie restriction, the ketogenic diet, and ketone bodies.  - (Maalouf, 2009)
  53. AMPK activation protects cells from oxidative stress‐induced senescence via autophagic flux restoration and intracellular NAD + elevation  - (Han, 2016)
  54. Regulation of AMP-activated protein kinase by natural and synthetic activators - (Hardie, 2015)
  55. Effects of Exhaustive Aerobic Exercise on Tryptophan-Kynurenine Metabolism in Trained Athletes  - (Strasser, 2016)
  56. PARP-1 inhibition increases mitochondrial metabolism through SIRT1 activation - (Bai, 2011)
  57. Carbohydrate restriction regulates the adaptive response to fasting - (Klein, 1992)
  58. Interventions to Slow Aging in Humans: Are We Ready? - (longo, 2015)
  59. Extending healthy life span–from yeast to humans - (longo, 2010)
  60. Dietary restriction with and without caloric restriction for healthy aging - (Lee, 2016)
  61. A Periodic Diet that Mimics Fasting Promotes Multi-System Regeneration, Enhanced Cognitive Performance, and Healthspan - (Longo, 2015)
  62. Diet mimicking fasting promotes regeneration and reduces autoimmunity and multiple sclerosis symptoms - (Longo, 2016)
  63. Resistance Exercise Training Alters Mitochondrial Function in Human Skeletal Muscle - (Porter, 2015)
  64. Ketogenic Diet Reduces Midlife Mortality and Improves Memory in Aging Mice  - (Newman, 2017)
  65. The NAD(+)/sirtuin pathway modulates longevity through activation of mitochondrial UPR and FOXO signaling - (Mouchiroud, 2013)
  66. NAMPT- mediated NAD(+) biosynthesis is essential for vision in mice - (Lin, 2016)
  67. NAD+ replenishment improves lifespan and healthspan in ataxia telangiectasia models via mitophagy and DNA repair - (Fang, 2016)
  68. Inhibiting poly ADP-ribosylation increases fatty acid oxidation and protects against fatty liver disease - (Gariani, 2017 )
  69. Interdependence of AMPK and SIRT1 for metabolic adaptation to fasting and exercise in skeletal muscle - (Canto, 2010)
  70. The NAD (+) precursor nicotinamide riboside enhances oxidative metabolism and protects against high-fat diet-induced obesity - (Canto, 2012)
  71. Nicotinamide riboside is uniquely and orally bioavailable in mice and humans - (Trammell, 2016)
  72. Nicotinamide riboside opposes type 2 diabetes and neuropathy in mice - (Trammell, 2016)
  73. Dietary leucine stimulates SIRT1 signaling through activation of AMPK - (Hongliang, 2012)
  74. Effective treatment of mitochondrial myopathy by nicotinamide riboside, a vitamin B3 - (Khan, 2014)
  75. NAD blocks high glucose induced mesangial hypertrophy via activation of the sirtuins-AMPK-mTOR pathway - (Zhuo, 2011)
  76. The effect of different exercise regimens on mitochondrial biogenesis and performance - (Philander, 2014)
  77. Dietary proanthocyanidins boost hepatic NAD+ metabolism and SIRT1 expression and activity in a dose-dependent manner in healthy rats - (Aragon’s, 2016)
  78. NAD+ Deficits in Age-Related Diseases and Cancer - (Garrido, 2017)
  79. Anti-diabetic and anti-lipidemic effects of chlorogenic acid are mediated by ampk activation - (Ong, 2013)
  80. Chlorogenic Acid Improves Late Diabetes through Adiponectin Receptor Signaling Pathways in db/db Mice - (Chang, 2015)
  81. Adenosine Monophosphate (AMP)-Activated Protein Kinase: A New Target for Nutraceutical Compounds - (Marin-Aguilar, 2017)
  82. The Effects of Ramadan Fasting on Body Composition, Blood Pressure, Glucose Metabolism, and Markers of Inflammation in NAFLD Patients: An Observational Trial - (Mazidi, 2014)
  83. Comparative effects of carbohydrate versus fat restriction on metabolic profiles, biomarkers of inflammation and oxidative stress in overweight patients with Type 2 diabetic and coronary heart disease: A randomized clinical trial. - (Raygan, 2016)
  84. Normal fasting plasma glucose and risk of type 2 diabetes diagnosis - (Nichols, 2008)
  85. Are We All Pre-Diabetic? - (Stokel,2016)
  86. Hepatic NAD+ deficiency as a therapeutic target for non-alcoholic fatty liver disease in aging - (Zhou, 2016)
  87. Effect of exercise intensity on post-exercise oxygen consumption and heart rate recovery - (Mann,2014)
  88. A 45-minute vigorous exercise bout increases metabolic rate for 14 hours - (Knab,2011)
  89. Effects of high-intensity resistance training on untrained older men. II. Muscle fiber characteristics and nuclei-cytoplasmic relationships - (Gerontol, 2000)
  90. Ketogenic Diet Reduces Midlife Mortality and Improves Memory in Aging Mice - (Newman, 2017)
  91. A Ketogenic Diet Extends Longevity and Healthspan in Adult Mice - (Roberts, 2017)
  92. NK cells link obesity-induced adipose stress to inflammation and insulin resistance - (Wensveen, 2015)
  93. The “Big Bang” in obese fat: Events initiating obesity-induced adipose tissue inflammation - (Wensveen, 2015)
  94. The impact of the Standard American Diet in rats: Effects on behavior, physiology and recovery from inflammatory injury - (Totsch, 2017)
  95. Bioenergetic state regulates innate inflammatory responses through the transcriptional co-repressor CtBP - (Shen, 2017)
  96. The Ketogenic Diet as a Treatment Paradigm for Diverse Neurological Disorders - (Stafstrom, 2012)
  97. Loss of NAD Homeostasis Leads to Progressive and Reversible Degeneration of Skeletal Muscle - (Fredrick 2016)
  98. Digestion and absorption of NAD by the small intestine of the rat - (Henderson, 1983)
  99. Effects of a wide range of dietary nicotinamide riboside (NR) concentrations on metabolic flexibility and white adipose tissue (WAT) of mice fed a mildly obesogenic diet - (Shi, 2017)
  100. Discoveries of nicotinamide riboside as a nutrient and conserved NRK genes establish a Preiss-Handler independent route to NAD+ in fungi and humans  - (Brenner, 2004)
  101. Nampt Expression Decreases Age-Related Senescence in Rat Bone Marrow Mesenchymal Stem Cells by Targeting Sirt1 - (Ma, 2017)

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